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The leukocyte infiltration into sites of infection is a fundamental host defense against invading pathogens where infiltrating leukocytes inactivate and clear the pathogens. Thus, inflammation is primarily a beneficial host response to noxious stimuli. However, inflammation acts as double-edged swords as an excessive and prolonged response can cause tissue damage, contributing to the pathogenesis of many disease states. Therefore, the response must be under the strict control of endogenous mechanism(s). We study the molecular mechanism(s) of inflammation and inflammatory response, which could be helpful for a new therapeutic strategy for the diseases that are hared to cure.